Artery Research

Volume 4, Issue 4, December 2010, Pages 168 - 168

P7.02 EFFECT OF CAPTOPRIL AND TELMISARTAN ON ANTI-CONTRACTILE PROPERTY OF PERIVASCULAR ADIPOSE TISSUE LOST AFTER HYPOXIA IN RAT MESENTERIC SMALL RESISTANCE ARTERIES

Authors
C. Agabiti Rosei1, A. Greenstein2, S. Withers2, A.M. Heagerty2
1Internal Medicine, University of Brescia, Italy
2Department of Medicine, Manchester University, United Kingdom
Available Online 2 December 2010.
DOI
10.1016/j.artres.2010.10.079How to use a DOI?
Open Access
This is an open access article distributed under the CC BY-NC license.

It has been previously demonstrated that inflammation in adipose tissue may be implicated in vascular dysfunction.Adipocytes secrete adiponectin, a physiological modulator of local vascular tone through an increased nitric oxide bioavailability.This capacity is lost in obesity by the development of adipocyte hypertrophy,leading to hypoxia,inflammation, and oxidative stress.

Aim: of the study was to investigate the feasibility a pharmacological modulation of the concerned effect,during hypoxia, in Wistar-Kyoto normotensive rats(WKY).

Materials and Methods: we investigated 25 WKY of 12 weeks of age.Mesenteric small resistance arteries were dissected and mounted on a wire myograph,according to Mulvany-Halpern technique(internal diameter about 200 μm).A concentration-response to norepinephrine(NE,from10−9to10−5Mol/l)was evaluated in the following conditions:1)in vessels with perivascular fat tissue(WF),2)in vessels without perivascular fat tissue(NoF);3)in WF vessels under hypoxic condition(WF+Hyp);4)in NoF vessels under hypoxic condition(NoF+Hyp);5)in WF+Hyp vessels incubated with telmisartan10−2Mol/l (WF+Hyp+Telm)or captopril10−2Mol/l for 3hour(WF+Hyp+Capt).

Results: Are summarized in the figure (active media stress: KPa, mean of two vessels for each rat).A significantly greater reactivity to NE was observed in NoF vessels compared with WF vessels(ANOVAp=0.003between curves).This increased reactivity is similar to that observed in WF+Hyp(ANOVAp= NS between, NoF and WF+Hyp).Captopril and Telmisartan were able to prevent the effect of hypoxia(ANOVA p<0.05 between WF+Hyp and WF+Hyp+Capt or Telm).

In conclusion the ACE inihibitor captopril and the angiotensin-receptor blocker telmisartan seem to be able to restore the anti-contractile effects of perivascular fat tissue lost after hypoxia, possibly through an inihibition of angiotensin II effects.

Journal
Artery Research
Volume-Issue
4 - 4
Pages
168 - 168
Publication Date
2010/12/02
ISSN (Online)
1876-4401
ISSN (Print)
1872-9312
DOI
10.1016/j.artres.2010.10.079How to use a DOI?
Open Access
This is an open access article distributed under the CC BY-NC license.

Cite this article

TY  - JOUR
AU  - C. Agabiti Rosei
AU  - A. Greenstein
AU  - S. Withers
AU  - A.M. Heagerty
PY  - 2010
DA  - 2010/12/02
TI  - P7.02 EFFECT OF CAPTOPRIL AND TELMISARTAN ON ANTI-CONTRACTILE PROPERTY OF PERIVASCULAR ADIPOSE TISSUE LOST AFTER HYPOXIA IN RAT MESENTERIC SMALL RESISTANCE ARTERIES
JO  - Artery Research
SP  - 168
EP  - 168
VL  - 4
IS  - 4
SN  - 1876-4401
UR  - https://doi.org/10.1016/j.artres.2010.10.079
DO  - 10.1016/j.artres.2010.10.079
ID  - AgabitiRosei2010
ER  -