Effect of Ginkgo Biloba Extract (EGb761) on Cerebral Nervous System in Patients with ALzheimer’s Disease
- DOI
- 10.2991/978-2-494069-31-2_33How to use a DOI?
- Keywords
- EGb761; AD; Aβ; AChE
- Abstract
Alzheimer's disease (AD) is a common neurodegenerative disease and a kind of dementia in the elderly. More and more studies have shown that using the standardized extract of Ginkgo biloba leaves (labeled EGb761) to intervene in the clinical treatment of AD patients can effectively improve their brain neuron damage. Although its biological mechanism and principle are still being explored. Among all kinds of research, the three most widely studied directions of scholars are: (i) EGb761 can prevent Aβ (1–42) oligomer induced results (ii) It can show anti neuroinflammatory toxicity (iii) It can produce natural acetylcholinesterase (AChE) inhibitors. The three directions simply analyze how EGb761 has a specific impact on the patient's brain nervous system after entering the human body. Based on the review of the above three directions, this paper will briefly discuss and review the specific effects of EGb761 on the brain nervous system of AD patients.
- Copyright
- © 2022 The Author(s)
- Open Access
- Open Access This chapter is licensed under the terms of the Creative Commons Attribution-NonCommercial 4.0 International License (http://creativecommons.org/licenses/by-nc/4.0/), which permits any noncommercial use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license and indicate if changes were made.
Cite this article
TY - CONF AU - Yanlei Gu PY - 2022 DA - 2022/12/29 TI - Effect of Ginkgo Biloba Extract (EGb761) on Cerebral Nervous System in Patients with ALzheimer’s Disease BT - Proceedings of the 2022 6th International Seminar on Education, Management and Social Sciences (ISEMSS 2022) PB - Atlantis Press SP - 267 EP - 273 SN - 2352-5398 UR - https://doi.org/10.2991/978-2-494069-31-2_33 DO - 10.2991/978-2-494069-31-2_33 ID - Gu2022 ER -